Abstract
The present research report has looked into the correlation between male infertility and the fluoride level in water, the occurrence of dental and skeletal fluorosis, [the volume and quality of] semen and the amount of fluoride contained therein. Results have shown that fluoride might be a leading cause of male infertility in regions with endemic fluorosis.
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Fluoride exposure arrests the acrosome formation during spermatogenesis via down-regulated Zpbp1, Spaca1 and Dpy19l2 expression in rat testes.
Highlights Fluoride alters the ultrastructure of acrosome and arrests acrosome formation during spermatogenesis. Fluoride down-regulates the expressions of ZPBP1, SPACA1 and Dpy19L2 during acrosome biogenesis in testis. Fluoride alters the ultrastructure of the nuclear lamina, and reduces LMNB2 expression in testis. The exposure and health effects of fluoride are an ongoing
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Developmental toxicity of sodium fluoride measured during multiple generations
Sodium fluoride (NaF) has been used to fluoridate drinking water in the United States since the mid 1940s. Because of the lack of reliable studies on the multigeneration effects of the compound, NaF (0, 25, 100, 175 or 250 ppm in drinking water) was given to rats continuously during three
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Fluoride impairs oocyte maturation and subsequent embryonic development in mice.
The damage caused by fluorosis is permanent, and has been recognized as a public health problem in a number of regions of the world. Although multiple studies provided evidence that sodium fluoride (NaF) elicits adverse effects on reproductive function, the effect of fluoride on female germ cell development is not
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Perinatal exposure to sodium fluoride with emphasis on territorial aggression, sexual behaviour and fertility in male rats.
Territorial aggression, sexual behaviour and fertility parameters were evaluated at adulthood of male rats previously exposed to different concentrations of sodium fluoride (Na-F) at their gestation, lactation and postweaning period till maturation. Sixty weanling male Wistar rats were received Na-F via their dams from second trimester of their pregnancy onward
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Amelioration of fluoride toxicity in some accessory reproductive glands and spermatozoa of rat
Sodium fluoride (NaF) at a dose of 10 mg/kg body weight was administered orally to male rats (Rettus norvegicus) daily for 30 and 50 days to evaluate the effect of the physiology of some sex accessory glands and sperm functions. The effects of withdrawal upon cessation of NaF ingestion, and
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