Industries that use fluoride-containing materials are usually aware of their toxicity and adopt adequate medical measures. It has been found that workers in factories who have not been considered as subjected to fluorine hazard and therefore have not been controlled had significantly elevated urinary fluoride levels. Most workers in a medically controlled fertilizer plant had normal urinary levels. Maintenance workers were found to have higher values, up to 12 mg/liter.
Skeletal fluorosis from brewed tea.
BACKGROUND: High fluoride ion (F(-)) levels are found in many surface and well waters. Drinking F(-)-contaminated water typically explains endemic skeletal fluorosis (SF). In some regions of Asia, however, poor quality "brick tea" also causes this disorder. The plant source of brick, black, green, orange pekoe, and oolong tea, Camellia
[Level of exposure to fluorides by the consumption of different types of milk in residents from an area of Mexico with endemic hydrofluorosis].
INTRODUCTION: Several studies have shown the presence of fluorosis (DF) in primary dentition, suggesting an exposure to fluorides (F-) in early childhood. Breast milk is recommended as an exclusive food until 6 months of age. Although it is mentioned that only a small amount of F- can be eliminated by
Fluoride exposure and thyroid function among adults living in Canada: Effect modification by iodine status.
Highlights Higher urinary fluoride levels are not associated with higher TSH levels in the general population of adults living in Canada. Iodine status modifies the relationship between urinary fluoride and TSH levels. Adults in Canada who have moderate-to-severe iodine deficiencies and higher urinary fluoride tend to have higher TSH
Evaluation of kidney injury biomarkers in an adult Mexican population environmentally exposed to fluoride and low arsenic levels.
Highlights Fluoride exposure increased renal injury biomarkers (ALB, Cys-C, KIM-1 and OPN). Fluoride could be considered an environmental kidney toxicant. Exposure to low concentrations of arsenic does not increase kidney injury biomarkers. Co-exposure to low arsenic level does not enhanced renal fluoride toxicity. Fluoride (F) is a toxicant widely distributed
A fatality due to ingestion of hydrofluoric acid.
We report a fatal case of hydrofluoric acid (HF) ingestion with suicidal intent. Quantitation using an ion-selective electrode for fluoride in fresh bile, gastric contents, kidney, liver, skeletal muscle, urine, and vitreous humor yielded 6.5, 39.0, 10.0, 6.0, 4.5, 5.0, and 4.5 ppm, respectively. In addition to the unfixed specimens,
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