Abstract
SUMMARY: In an extension of previous work on fluoride (F) toxicity in a group of 80 Swiss albino mice, the mitigating effects of polyphenols in black tea on the F-induced increase in glycogen, cholesterol, and total lipids in the cerebral hemisphere (CH), cerebellum (CB), and medulla oblongata (MO) regions of the brain were studied. Oral administration of sodium fluoride (6 and 12 mg NaF/kg bw/day) to the mice for 30 days resulted in a significant dose-dependent increase in the parameters studied. Withdrawal of treatment for 30 days caused significant but only partial recovery. Administration of a 2% black tea infusion alone for 30 days did not cause any significant alteration. However, concurrent administration of NaF and black tea infusion for 30 days produced significant mitigation in all the parameters.
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Black tea extract mitigation of NaF-induced lipid peroxidation in different regions of mice brains.
SUMMARY: As part of our investigation of fluoride toxicity effects in a group of 80 Swiss albino adult male mice, we examined the mitigating effects of black tea extract (BTE) on the F-induced enzymatic and non-enzymatic parameters of oxidative stress in the cerebral hemisphere (CH), cerebellum (CB), and medulla oblongata (MO) of the
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Public-health risks from tea drinking: Fluoride exposure.
Aims: Due to new evidence on fluoride neurotoxicity during early life, this study examined maternal exposure to fluoride through tea consumption in a low-fluoride region and measured fluoride releases from commercially available teas (tea bags and loose teas) to determine the need to limit fluoride exposure. Methods:
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Amelioration by black tea of sodium fluoride-induced changes in protein content of cerebral hemisphere, cerebellum and medulla oblongata in brain region of mice.
Oral administration of sodium fluoride (NaF, 6 and 12 mg/kg body weight/day) to Swiss strain male albino mice for 30 days caused significant dose-dependant reduction in the content of acidic, basic, neutral, and total protein in cerebral hemisphere, cerebellum and medulla oblongata region of brain. After 30 days of NaF
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Analysis of naturally occurring fluoride in commercial teas and estimation of its daily intake through tea consumption
The aim of this study was to determine the levels of fluoride in commercial teas and to estimate the contribution of tea consumption to the fluoride recommended daily allowance. A total of 558 tea products in 6 categories, green tea, black tea, oolong tea, pu'er tea, white tea, and reprocessed
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Black tea amelioration of sodium fluoride-induced alterations of DNA, RNA, and protein contents in the cerebral hemisphere, cerebellum, and medulla oblongata regions of mouse brain.
Summary: Oral administration of sodium fluoride (NaF, 6 and 12 mg/kg body weight/day) to Swiss male albino mice for 30 days caused significant, dose-dependent reduction in DNA, RNA, and protein contents in cerebral hemisphere, cerebellum, and medulla oblongata of the brain. After 30 days of NaF treatment, followed by withdrawal
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Fluoride: Developmental Neurotoxicity.
Developmental Neurotoxicity There has been a tremendous amount of research done on the association of exposure to fluoride with developmental neurotoxicity. There are over 60 studies reporting reduced IQ in children and several on the impaired learning/memory in animals. And there are studies which link fluoride to Attention Deficit Hyperactivity Disorder. Teaching
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Skeletal Fluorosis in the U.S.
Although there has been a notable absence of systematic studies on skeletal fluorosis in the U.S., the available evidence indicates that the consumption of artificially fluoridated water is likely to cause skeletal fluorosis and other forms of bone disease in people with kidney disease and other vulnerable populations.
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Fluoride's Effect on Fetal Brain
The human placenta does not prevent the passage of fluoride from a pregnant mother's bloodstream to the fetus. As a result, a fetus can be harmed by fluoride ingested pregnancy. Based on research from China, the fetal brain is one of the organs susceptible to fluoride poisoning. As highlighted by the excerpts
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Estimated "Threshold" Doses for Skeletal Fluorosis
For over 40 years health authorities stated that in order to develop crippling skeletal fluorosis, one would need to ingest between 20 and 80 mg of fluoride per day for at least 10 or 20 years. This belief, however, which played an instrumental role in shaping current fluoride policies, is now acknowledged by the National Academy of Sciences (NAS) and other US health authorities to be incorrect.
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Fluoride's Direct Effects on Brain: Animal Studies
The possibility that fluoride ingestion may impair intelligence and other indices of neurological function is supported by a vast body of animal research, including over 40 studies that have investigated fluoride's effects on brain quality in animals. As discussed by the National Research Council, the studies have consistently demonstrated that fluoride, at widely varying concentrations, is toxic to the brain.
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