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  • [Investigation of children’s intelligence quotient and dental fluorosis in drinking water-type of endemic fluorosis area in Pucheng county Shaanxi province before and after drinking water change].

    Objective: To further understand the effect of anti—fluoride water on intellectual development of children and dental fluorosis in drinking water-type of endemic fluorosis area Pucheng county Shaanxi province. Methods: Two hundred 8 to 12 years old children were randomly selected in endemic fluorosis area with changed water or unchanged water, in

  • Protective effect of ascorbic acid and Ginkgo biloba against learning and memory deficits caused by fluoride.

    Fluoride is present in the ground water, World Health Organization permitted level of fluoride in the ground water is 0.5 ppm. Tooth pastes, mouth washes, tea and sea fish are the sources of fluoride. Exposure to these multiple sources results in several adverse effects in addition to the fluorosis. The

  • Potentials for developmental fluoride neurotoxicity

    The foetus and the child are particularly vulnerable to pollution. The foetus shares the mother’s exposure and accumulated body burden of pollutants, and some chemicals are transferred to the infant via human milk. Occurrence of severe dental fluorosis in a child, whose mother had worked at the Danish cryolite factory

  • Histological changes in the brain of young fluoride-intoxicated rats

    Wistar albino rats were exposed to 30 or 100 ppm fluoride (as NaF) in drinking water during their fetal, weanling, and post-weaning stages until the age of ten weeks. Rats exposed to 30 ppm fluoride did not show any notable alterations in brain histology, whereas rats exposed to 100 ppm

  • Proteomic analysis of hippocampus in offspring male mice exposed to fluoride and lead

    Fluoride and lead are two common pollutants in the environment. Previous investigations have found that high fluoride exposure can increase the lead burden. In this experiment, in order to study on the molecular mechanisms of central neural system injury induced by the above two elements, differently expressed protein spots in

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