Study Tracker

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Alterations in epididymal proteomics and antioxidant activity of mice exposed to fluoride.

January 1, 2018 | Archives of Toxicology | Sun Z, Li S, Yu Y, Chen H, Ommati MM, Manthari RK, Niu R, Wang J. | 92(1):169-180.

Fluoride-Induced Autophagy via the Regulation of Phosphorylation of Mammalian Targets of Rapamycin in Mice Leydig Cells.

October 11, 2017 | Journal of Agricultural and Food Chemistry | Zhang J, Zhu Y, Shi Y, Han Y, Liang C, Feng Z, Zheng H, Eng M, Wang J. | 65(40):8966-8976.

Sodium fluoride exposure exerts toxic effects on porcine oocyte maturation.

December 6, 2017 | Scientific Reports | Liang S, Nie ZW, Zhao M, Niu YJ, Shin KT, Cui XS. | 7(1):17082

Vitamin E and Lycopene Reduce Coal Burning Fluorosis-induced Spermatogenic Cell Apoptosis via Oxidative Stress-mediated JNK and ERK Signaling Pathways.

December 22, 2017 | Bioscience Reports | Tian Y, Xiao Y, Wang B, Sun C, Tang K, Sun F. | [Epub ahead of print]

Sodium fluoride disturbs DNA methylation of NNAT and declines oocyte quality by impairing glucose transport in porcine oocytes.

December 29, 2017 | Environmental and Molecular Mutagenesis | Liu X, Nie ZW, Gao YY, Chen L, Yin SY, Zhang X, Hao C, Miao YL. | [Epub ahead of print]

Impact of subchronic exposure to triclosan and/or fluoride on estrogenic activity in immature female rats: The expression pattern of calbindin-D9k and estrogen receptor a genes.

January 11, 2018 | Journal of Biochemical and Molecular Toxicology. | Abd-Elhakim YM, Mohammed AT, Ali HA. | [Epub ahead of print]

The protective properties of selected naturally occurring antioxidants of plant origin against fluoride-induced neurotoxicity.

April-June 2017 | Fluoride | Goschorska M, Giewon K, Hasior N, Gutowska I, Baranowska-Bosiacka I, Rac ME, Chulbek D. | 50(2):203–212.

Attenuating effect of Vitamin E on the deficit of learning and memory of rats with chronic fluorosis: the mechanism may involve muscarinic acetylcholine receptors.

July-September 2017 | Fluoride | Dong YT, Wei N, Qi XL, Liu XH, Chen D, Zeng XX, Guan ZZ. | 50(3):354–364.

The Effect of Chronic Fluorosis on Calcium Ions and CaMKIIa, and c-fos Expression in the Rat Hippocampus.

April 2018 | Biological Trace Element Research | Teng Y, Zhang J, Zhang Z, Feng J. | 182(2):295-302.

Effects of Calcium on Drinking Fluorosis-induced Hippocampal Synaptic Plasticity Impairment in the Offspring of Rats.

December 29, 2017 | Translational Neuroscience | Zigu Z, Xiaoyu W, Weiwei N, Qiuxia L, Rui Z, Wei O. | 8:191-200.

Research Studies

Study Tracker

Study Tracker

The most up-to-date and comprehensive source for studies on fluoride's multifaceted effects on human and animal health, with 100+ years of research organized by health category, type of study, author, journal, and date.

Health Category:

Health Sub-Category:

Study Type:

Publish Date:

-

Alterations in epididymal proteomics and antioxidant activity of mice exposed to fluoride.

Fluoride-Induced Autophagy via the Regulation of Phosphorylation of Mammalian Targets of Rapamycin in Mice Leydig Cells.

Sodium fluoride exposure exerts toxic effects on porcine oocyte maturation.

Vitamin E and Lycopene Reduce Coal Burning Fluorosis-induced Spermatogenic Cell Apoptosis via Oxidative Stress-mediated JNK and ERK Signaling Pathways.

Sodium fluoride disturbs DNA methylation of NNAT and declines oocyte quality by impairing glucose transport in porcine oocytes.

Impact of subchronic exposure to triclosan and/or fluoride on estrogenic activity in immature female rats: The expression pattern of calbindin-D9k and estrogen receptor a genes.

The protective properties of selected naturally occurring antioxidants of plant origin against fluoride-induced neurotoxicity.

Attenuating effect of Vitamin E on the deficit of learning and memory of rats with chronic fluorosis: the mechanism may involve muscarinic acetylcholine receptors.

The Effect of Chronic Fluorosis on Calcium Ions and CaMKIIa, and c-fos Expression in the Rat Hippocampus.

Effects of Calcium on Drinking Fluorosis-induced Hippocampal Synaptic Plasticity Impairment in the Offspring of Rats.