Diagnosis of professional fluorosis (PF) and its classification have been poorly studied. After development of PF classification by D. M. Zislin and E. Ya. Girskaya [3], new study methods have been introduced in clinical practice which have allowed some indications of the disease to be revealed [2, 5, 6, 8]. However, this classification does not take etiology into account. There are no parameters for fluorine in the human biological environment. Both domestic and foreign authors have confirmed t
Deforming osteoarthrosis (DOA) has the highest prevalence rate among joint diseases [1, 3-9]. There is little information about the development of DOA in subjects in contact with fluoride compounds [2].
We have studied the characteristics of the diagnosis and treatment of DOA with patients who had previous contact with fluoride compounds in comparison to primary DOA. 484 patients have been examined, of them 106 had primary DOA, and 378 were workers in the main professions of electrolysis depa
Fluorine is a poison with a broad spectrum of action affecting the metabolic processes, tissue respiration, and neuroendocrine regulation. For this reason, even before affecting the bones, occupational fluorosis manifests itself clinically as a generalized disease with syndromes affecting a number of important systems and organs, including the nervous system.
The neurotoxic effect of fluorine compounds has been proven by numerous experimental studies (A. F. Aksyuk and G. V. Bulychev; S. K. Bi
Our team performed investigations in Yuqing Village and Guanfang Village (1) which were seriously affected by endemic fluorosis, from September to October 1985; those two villages are only 1,500 meters distant from Mangbu District of Zhenxiong County, and the life and production conditions in these two villages are similar to those in Mangbu District, only that these two villages are heavily polluted by coal smoke from daily use. We performed a total of 292 electrocardiograms (ECGs) on villagers
