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Co-exposure to non-toxic levels of cadmium and fluoride induces hepatotoxicity in rats via triggering mitochondrial oxidative damage, apoptosis, and NF-kB pathways.

References Abarikwu SO, Njoku R-C, Lawrence CJ, Charles IA, Ikewuchi JC (2017) Rutin ameliorates oxidative stress and preserves hepatic and renal functions following exposure to cadmium and ethanol. Pharm Biol 55:2161–2169 CAS Article Google Scholar Abu-El-Zahab HS, Hamza RZ, Montaser MM, El-Mahdi MM, Al-Harthi WA (2019) Antioxidant, antiapoptotic, antigenotoxic, and hepatic ameliorative effects of L-carnitine and selenium on cadmium-induced hepatotoxicity

Assessment of phytotoxicity of leachates from landfilled waste and dust from foundry.

In contrast to chlorides and sulfates, fluorides are toxic to biota even at low concentrations (Stevens et al. 2000; Palmieri et al. 2014). Fluoride concentration was analyzed in the leachate, as it may be present in foundry waste. Organic binders are the main source of fluorides in foundry waste leachate (http://life-foundrysand.com). The fluoride concentration was low in all tested leachate. Plant resistance to fluoride depends on the plant species. Stevens et al. (1998) report the toxic for p