Excerpt:

The present study was carried out during May 2010 to December 2011 in three villages which were randomly selected from Warora tehsil of Chandrapur district which is one of the endemic district of Maharashtra. . . . All the presently available ground water samples were collected and the mean fluoride concentration  was estimated by the ion analyser method, varied from 0.73 to 2.80 mg/L compared to the recommended  maximum permissible limit of 1.5 mg/L.

As seen in Table 5 a high prevalence of  skeletal fluorosis was observed from habitual users of black tea and tobacco [11.91% – 19.85%] whereas a relatively very low  prevalence [3.72% – 7.1%] of fluorosis was observed among non-users. . . .  Over 90% of the people affected with severe fluorosis belongs to the low socio-economic status and they had generalized nutritional deficiencies.

In the present clinical examination it was observed that the most common symptoms joint pain (35.75%), back pain (29.01%) and other symptoms like tingling and numbing of extremities, shoulder pain, neck pain and stiffness are the symptoms of skeletal fluorosis. Earlier investigators found that back pain (68%), joint pain (22%), pain or stiffness in the backbone and hip (16.2%) and pain or stiffness in joints and backbone  (15.5%) and back pain (73%) and neck pain (34%). The common symptoms are pain in the neck and back  associated with rigidity, joint pains and paraesthesia of the limb.

Print PDF

• Related Studies :

  • Non-Endemic Skeletal Fluorosis: Causes And Associated Secondary Hyperparathyroidism (Case Report and Literature Review).

    Highlights Fluorocarbon “huffing” is an under-appreciated cause of skeletal fluorosis (SF) We present a SF case with hyperparathyroidism, osteosclerosis, and osteomalacia SF may go undetected due to variation in symptoms, radiology, and biochemistry Dietary calcium, prior bone health, and skeletal F exposure influence SF features SF is common in

  • Skeletal fluorosis and instant tea

    Tea drinking remains popular in the United States and increasingly is suggested to promote health. We caution that skeletal fluorosis can result from consumption of excessive amounts of instant tea because of substantial fluoride levels in some commercial preparations. Case report A 52-year-old white woman consulted in 1998 for dense lumbar vertebras

  • Fluoride exposure and bone status in patients with chronic intestinal failure who are receiving home parenteral nutrition

    BACKGROUND AND OBJECTIVE:  Metabolic bone disease is frequent in chronic intestinal failure. Because fluoride has a major effect on bones, the status of both fluoride and bone was studied in long-term home parenteral nutrition (HPN) patients. DESIGN:  We studied 31 adults aged (x +/- SD) 56.3 +/- 15.1 y, mainly

  • Skeletal fluorosis mimicking seronegative spondyloarthropathy: a deceptive presentation

    Skeletal fluorosis is rarely recognized early and is amajor cause of morbidity.We report on a 40-year-old man with skeletal fluorosis mimicking seronegative spondyloarthropathy.

  • Fluoride-related bone disease associated with habitual tea consumption

    Acquired osteosclerosis is a rare disorder of bone formation but an important consideration in adults with sclerotic bones or elevated bone density results. In such patients, malignancy, hepatitis C, and fluorosis should all be considered when making a diagnosis. We describe 4 patients evaluated at our Metabolic Bone Disease Clinic

• Related FAN Content :

  • Mayo Clinic: Fluoridation & Bone Disease in Renal Patients

    The available evidence suggests that some patients wtih long-term renal failure are being affected by drinking water with as little as 2 ppm fluoride. The finding of adverse effects in patients drinking water with 2 ppm of fluoride suggests that a few similar cases may be found in patients imbibing 1 ppm, especially if large volumes are consumed, or in heavy tea drinkers. The finding of adverse effects in patients drinking water with 2 ppm of fluoride suggests that a few similar cases may be found in patients imbibing 1 ppm, especially if large volumes are consumed, or in heavy tea drinkers and if fluoride is indeed the cause. It would seem prudent, therefore, to monitor the fluoride intake of patients with renal failure living in high fluoride areas.

  • Fluoridation, Dialysis & Osteomalacia

    In the 1960s and 1970s, doctors discovered that patients receiving kidney dialysis were accumulating very high levels of fluoride in their bones and blood, and that this exposure was associated with severe forms of osteomalacia, a bone-softening disease that leads to weak bones and often excruciating bone pain. Based on

  • "Pre-Skeletal" Fluorosis

    As demonstrated by the studies below, skeletal fluorosis may produce adverse symptoms, including arthritic pains, clinical osteoarthritis, gastrointestinal disturbances, and bone fragility, before the classic bone change of fluorosis (i.e., osteosclerosis in the spine and pelvis) is detectable by x-ray. Relying on x-rays, therefore, to diagnosis skeletal fluorosis will invariably fail to protect those individuals who are suffering from the pre-skeletal phase of the disease. Moreover, some individuals with clinical skeletal fluorosis will not develop an increase in bone density, let alone osteosclerosis, of the spine. Thus, relying on unusual increases in spinal bone density will under-detect the rate of skeletal fluoride poisoning in a population.

  • Kidney Patients Are at Increased Risk of Fluoride Poisoning

    It is well established that individuals with kidney disease are susceptible to suffering bone damage and other ill effects from low levels of fluoride exposure. Kidney patients are at elevated risk because when kidneys are damaged they are unable to efficiently excrete fluoride from the body. As a result, kidney patients

  • Fluoride & Osteoarthritis

    While the osteoarthritic effects that occurred from fluoride exposure were once considered to be limited to those with skeletal fluorosis, recent research shows that fluoride can cause osteoarthritis in the absence of traditionally defined fluorosis. Conventional methods used for detecting skeletal fluorosis, therefore, will fail to detect the full range of people suffering from fluoride-induced osteoarthritis.