Abstract
Three hundred seventy-four 12- to 14-year-old children were examined to determine modified tooth surface index of fluorosis (TSIF) scores, and to assess the association among fluoride enamel opacities and water fluoride levels, preschool dietary patterns, and dentifrice consumption. The subjects included boys and girls of both black and white races who reported life-long residence in either Augusta or adjoining Richmond County. “City” children had communal water consistently fluoridated at levels of 0.9 to 1.2 ppm. “County” children had water variably fluoridated at levels of 0.2 to 0.9 ppm. TSIF scores were recorded on each included tooth and the highest TSIF tooth score was noted for each subject. The frequency of TSIF scores in all subjects was analyzed for dental arch symmetry and for association with city/county residence. The frequency of TSIF scores then was analyzed separately for “city” children and “county” children for an association with race, gender, preschool dietary habits, and dentifrice ingestion. Chi-square analysis revealed that higher TSIF scores were associated with city children significantly more than with county children. There was no association of TSIF scores either in the city children or the county children with respect to gender, race, preschool dietary patterns, or dentifrice ingestion. TSIF scores were bilaterally symmetrical, but were higher in the maxillary arch than in the mandibular arch.
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Low birth weight and dental fluorosis: is there an association?
OBJECTIVE: The association between low birth weight and dental fluorosis was explored in a cross-sectional study to explain the higher prevalence of dental fluorosis among African-American children. METHODS: Birth weight data on 960 children were obtained from the New York State Birth Registry. Data on race, fluoride exposure, sociodemographic characteristics, and
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Prevalence of dental mottling in school-aged lifetime residents of 16 Texas communities
The severity of dental mottling in 2,592 school-aged, lifetime residents of 16 Texas communities was investigated in 1980-81 to identify factors associated with mottling and to construct a prediction model for the prevalence of mottling. The communities were selected to obtain a wide range of levels of fluoride in the
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The role of partial recording protocols in reporting prevalence and severity of dental fluorosis
OBJECTIVES: To evaluate the role of partial recording protocols (PRPs) in reporting prevalence and severity of dental fluorosis and assess whether prevalence/severity estimates derived from PRPs differ by race/ethnicity. METHODS: Data from the National Health and Nutrition Examination Survey (NHANES) for the years 1999-2004 were analyzed with Stata® v.11. Prevalence of
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Differences in exposure and biological markers of fluoride among White and African American children
OBJECTIVE: To determine differences in self-reported fluoride exposure and fluoride exposure biomarkers between two racial groups. METHODS: Questionnaires regarding fluoride exposure, urine and water collection kits were distributed to African American and White 7-14-year-old children. Children received a dental exam for fluorosis. Water, urine, and saliva were analyzed for fluoride content.
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Fluoride exposure and dental fluorosis in Newburgh and Kingston, New York: policy implications
OBJECTIVES: This analysis was conducted to determine the changes in the effect of exposure to fluoridation and other sources of fluoride on dental fluorosis in children attending Newburgh and Kingston school districts in New York State. METHODS: Data for this analysis were obtained from two surveys conducted in the 1986 and
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Community Fluorosis Index (CFI)
The current Community Fluorosis Index for U.S. adolescents as a whole (from both fluoridated and non-fluoridated areas) is roughly 5 times higher than the CFI health authorities predicted for fluoridated areas when fluoridation first began. It is also higher than the CFI that the NIDR found in fluoridated areas back in the 1980s. It is readily apparent, therefore, that children are ingesting far more fluoride than was the case in the 1950s, and even as recently as the 1980s.
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Dental Fluorosis in the U.S. 1950-2004
Before the widespread use of fluoride in dentistry, dental fluorosis was rarely found in western countries. Today, with virtually every toothpaste now containing fluoride, and most U.S. water supplies containing fluoride chemicals, dental fluorosis rates have reached unprecedented levels. In the 1950s, it was estimated that only 10% of children in
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Nutrient Deficiencies Enhance Fluoride Toxicity
It has been known since the 1930s that poor nutrition enhances the toxicity of fluoride. As discussed below, nutrient deficiencies have been specifically linked to increased susceptibility to fluoride-induced tooth damage (dental fluorosis), bone damage (osteomalacia), neurotoxicity (reduced intelligence), and mutagenicity. The nutrients of primary importance appear to be calcium,
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Skeletal Fluorosis & Individual Variability
One of the common fallacies in the research on skeletal fluorosis is the notion that there is a uniform level of fluoride that is safe for everyone in the population. These "safety thresholds" have been expressed in terms of (a) bone fluoride content, (b) daily dose, (c) water fluoride level, (d) urinary fluoride level, and (e) blood fluoride level. The central fallacy with each of these alleged safety thresholds, however, is that they ignore the wide range of individual susceptibility in how people respond to toxic substances, including fluoride.
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Mayo Clinic: Fluoridation & Bone Disease in Renal Patients
The available evidence suggests that some patients wtih long-term renal failure are being affected by drinking water with as little as 2 ppm fluoride. The finding of adverse effects in patients drinking water with 2 ppm of fluoride suggests that a few similar cases may be found in patients imbibing 1 ppm, especially if large volumes are consumed, or in heavy tea drinkers. The finding of adverse effects in patients drinking water with 2 ppm of fluoride suggests that a few similar cases may be found in patients imbibing 1 ppm, especially if large volumes are consumed, or in heavy tea drinkers and if fluoride is indeed the cause. It would seem prudent, therefore, to monitor the fluoride intake of patients with renal failure living in high fluoride areas.
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