SUMMARY: As part of our investigation of fluoride toxicity effects in a group of 80
Swiss albino adult male mice, we examined the mitigating effects of black tea extract
(BTE) on the F-induced enzymatic and non-enzymatic parameters of oxidative stress
in the cerebral hemisphere (CH), cerebellum (CB), and medulla oblongata (MO) of the brains of these mice. Oral administration of 6 and 12 mg NaF/kg bw/day to the mice for 30 days resulted in a significant increase in level of lipid peroxidation (LPO) and dehydroascorbic (DAA) acid as well as a decrease in glutathione (GSH), total
ascorbic acid (TAA), and reduced ascorbic acid. In addition, the activities of the
enzymatic antioxidants catalase (CAT), superoxide dismutase (SOD), glutathione
reductase (GSH-Pr), and glutathione peroxidase (GSH-Px) as well as cholinesterase
(ChE) also decreased. No significant recovery in any of these parameters was
observed upon withdrawal of the NaF treatment for 30 days. However, administration of BTE along with the NaF during the experiment resulted in significant mitigation of all the NaF-induced effects that were examined.
Amelioration by black tea of sodium fluoride-induced changes in protein content of cerebral hemisphere, cerebellum and medulla oblongata in brain region of mice.
Oral administration of sodium fluoride (NaF, 6 and 12 mg/kg body weight/day) to Swiss strain male albino mice for 30 days caused significant dose-dependant reduction in the content of acidic, basic, neutral, and total protein in cerebral hemisphere, cerebellum and medulla oblongata region of brain. After 30 days of NaF
Mitigation of sodium fluoride induced toxicity in mice brain by black tea infusion.
SUMMARY: In an extension of previous work on fluoride (F) toxicity in a group of 80 Swiss albino mice, the mitigating effects of polyphenols in black tea on the F-induced increase in glycogen, cholesterol, and total lipids in the cerebral hemisphere (CH), cerebellum (CB), and medulla oblongata (MO) regions of
Fluoride concentration and pH of iced tea products
The objective of this study was to determine the fluoride concentration and pH of 44 iced tea samples and hence to assess the possible role of these beverages as systemic fluoride source as well as their potential cariogenic and erosive character. Ten tea samples were available as a granular instant powder, and 34 as
Fluoride content in caffeinated, decaffeinated and herbal teas
The fluoride contents of infusions prepared from 44 different brands and types of teas were measured. Fluoride concentrations ranged from 0.34 to 3.71 ppm (mean = 1.50 ppm) in caffeinated tea infusions, 0.02-0.14 ppm (mean = 0.05 ppm) in herbal tea infusions, and 1.01-5.20 ppm (mean = 3.19) in decaffeinated tea infusions. This is
Fluoride in tea from Shandong Province, China and exposure assessment.
In this study fluoride in 170 tea samples from Shandong province (China) was determined using a selective ion electrode. The fluoride concentrations ranged from 31.2 to 338 mg/kg with an average of 119 mg/kg. The mean fluoride concentrations were 164, 121, 98.7 and 96.8 mg/kg, which corresponded to Pu'erh tea,
Related Studies :
NRC (2006): Fluoride's Neurotoxicity and Neurobehavioral Effects
The NRC's analysis on fluoride and the brain.
The Lancet: Fluoride Studies in a Patient with Arthritis
It is possible that fluoride intake from tea may be sufficient to cause fluorosis, and I report here a case which gives some evidence for this.
Fluoride's Effect on Fetal Brain
The human placenta does not prevent the passage of fluoride from a pregnant mother's bloodstream to the fetus. As a result, a fetus can be harmed by fluoride ingested pregnancy. Based on research from China, the fetal brain is one of the organs susceptible to fluoride poisoning. As highlighted by the excerpts
Estimated "Threshold" Doses for Skeletal Fluorosis
For over 40 years health authorities stated that in order to develop crippling skeletal fluorosis, one would need to ingest between 20 and 80 mg of fluoride per day for at least 10 or 20 years. This belief, however, which played an instrumental role in shaping current fluoride policies, is now acknowledged by the National Academy of Sciences (NAS) and other US health authorities to be incorrect.
Skeletal Fluorosis in the U.S.
Although there has been a notable absence of systematic studies on skeletal fluorosis in the U.S., the available evidence indicates that the consumption of artificially fluoridated water is likely to cause skeletal fluorosis and other forms of bone disease in people with kidney disease and other vulnerable populations.
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