Abstract
X-ray examinations of 98 potroom workers at an aluminum refinery plant in China have been carried out to study skeletal disorders that might be related to f-exposure. The examinations included the pelvis, lumbar vertebrae, radius, ulna, tibia, and fibula. Changes in skeletal system, such as bone density and trabeculae structure, the appearance of osteophytes and exostosis, and the calcification of interosseous membranes and ligaments were assessed by two orthopedic surgeons with the double blind test. NO cases of typical skeletal fluorosis were found among the study group, but the appearance of lumbar vertebral osteophyte in the 45-54 year group, exposed to fluoride for more than 20 years, was significantly more frequent than that in the respective control groups No significant differences in other aspects of osteosclerosis between the F-exposed and the control groups were observed.
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The impact of fluoride in drinking water on oral health and skeletal system of school children
Modern life styles even among people in rural areas have created an increased demand for dental cosmetology. Dental fluorosis due to its cosmetic effect gains more public health importance today. In the scenario of increasing awareness of environmental health hazards, among people, the research into the biology of fluorosis conducted
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Impacts of soil and water fluoride contamination on the safety and productivity of food and feed crops: A systematic review.
Highlights Plants tend to accumulate fluoride mostly in the root system Accumulation of F in plant tissues is dose-dependent with some exceptions F contamination of food crops can represents an actual health hazard in polluted areas F can alter chlorophyll levels, plant physiology and can induce oxidative stress Evidences of
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Calcium alleviates fluoride-induced bone damage by inhibiting endoplasmic reticulum stress and mitochondrial dysfunction.
Excessive fluoride mainly causes skeletal lesions. Recently, it has been reported that an appropriate level of calcium can alleviate fluorosis. However, the appropriate concentration and mechanism of calcium addition is unclear. Hence, we evaluated the histopathology and ultrastructure, DNA fragmentation, hormonal imbalances, biomechanical levels and the expression of apoptosis-related genes,
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Fluoride osteosclerosis
1. Twenty-three cases of fluoride osteosclerosis are presented. 2. These cases were gathered from 170,000 roentgenographic examinations of the spine and pelvis of patients living in Texas and Oklahoma where many communities have excessive fluoride content in their drinking water. 3. In each case, adequate clinical examination failed to establish any relationship
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A Brief and Critical Review of Chronic Fluoride Poisoning (Fluorosis) in Domesticated Water Buff aloes (Bubalus bubalis) in India: Focus on its Impact on Rural Economy.
In the rural areas of India, fl uoridated drinking water, industrial fluoride pollution and fluoride rich feed phosphate supplements are the major sources of fluoride exposure for domesticated water buffaloes (Bubalus bubalis). However, the fluoridated drinking groundwater is the commonest and principal source of fluoride exposure for these ruminants. Chronic
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Fluoride & Osteoarthritis
While the osteoarthritic effects that occurred from fluoride exposure were once considered to be limited to those with skeletal fluorosis, recent research shows that fluoride can cause osteoarthritis in the absence of traditionally defined fluorosis. Conventional methods used for detecting skeletal fluorosis, therefore, will fail to detect the full range of people suffering from fluoride-induced osteoarthritis.
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Skeletal Fluorosis: The Misdiagnosis Problem
It is a virtual certainty that there are individuals in the general population unknowingly suffering from some form of skeletal fluorosis as a result of a doctor's failure to consider fluoride as a cause of their symptoms. Proof that this is the case can be found in the following case reports of skeletal fluorosis written by doctors in the U.S. and other western countries. As can be seen, a consistent feature of these reports is that fluorosis patients--even those with crippling skeletal fluorosis--are misdiagnosed for years by multiple teams of doctors who routinely fail to consider fluoride as a possible cause of their disease.
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"Pre-Skeletal" Fluorosis
As demonstrated by the studies below, skeletal fluorosis may produce adverse symptoms, including arthritic pains, clinical osteoarthritis, gastrointestinal disturbances, and bone fragility, before the classic bone change of fluorosis (i.e., osteosclerosis in the spine and pelvis) is detectable by x-ray. Relying on x-rays, therefore, to diagnosis skeletal fluorosis will invariably fail to protect those individuals who are suffering from the pre-skeletal phase of the disease. Moreover, some individuals with clinical skeletal fluorosis will not develop an increase in bone density, let alone osteosclerosis, of the spine. Thus, relying on unusual increases in spinal bone density will under-detect the rate of skeletal fluoride poisoning in a population.
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