Abstract
INTRODUCTION:
Fluoride (F) is a toxic and reactive element, and exposure to it passes almost unnoticed, with the consumption of tea, fish, meat, fruits, etcetera and articles of common use such as: toothpaste additives; dental gels, non-stick pans and razor blades as Teflon. It has also been used with the intention of reducing the dental cares.
DEVELOPMENT:
Fluoride can accumulate in the body, and it has been shown that continuous exposure to it causes damaging effects on body tissues, particularly the nervous system directly without any previous physical malformations.
BACKGROUND:
Several clinical and experimental studies have reported that the F induces changes in cerebral morphology and biochemistry that affect the neurological development of individuals as well as cognitive processes, such as learning and memory. F can be toxic by ingesting one part per million (ppm), and the effects they are not immediate, as they can take 20 years or more to become evident.
CONCLUSION:
The prolonged ingestion of F may cause significant damage to health and particularly to the nervous system. Therefore, it is important to be aware of this serious problem and avoid the use of toothpaste and items that contain F, particularly in children as they are more susceptible to the toxic effects of F.
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Effects of high fluoride and arsenic on brain biochemical indexes and learning-memory in rats
Nine-six Wistar rats were randomly divided into four groups of 24 rats in each group (female:male = 1:1). Over a period up to 90 days, with one untreated group as controls, the other three groups were administered, respectively, high fluoride (100 mg NaF/L), high arsenic (50 mg As2O3/L), or both
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Cerebral neurodegeneration in experimental fluorosis
Sprague Dawley albino rats were treated with 30, 45, and 75 mg NaF/kg body weight/day respectively for 20 days and 35 days to study neurotoxic effect of fluoride. The control rats were injected with double distilled water 1cc/kg body weight/day. The animals were sacrificed and the cerebrum was analysed for
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Effect of sodium fluoride on the cerebellar cortex of adult albino rats and the possible protective role of vitamin B6: a light and electron microscopic study
Introduction: Sodium fluoride (NaF) toxicity has been a subject of extensive studies over the last years because of their environmental persistence and world widespread distribution. Its toxicity on many organs has received considerable attention, but its neurotoxicity has not been entirely studied. Aim of the Work: Studying the histological changes occurring
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Challenges of fluoride pollution in environment: Mechanisms and pathological significance of toxicity – A review.
Highlights Fluoride exposure induces mitochondrial function dysfunction. Fluoride exposure mediates organs injury involved in apoptosis and autophagy. Fluoride exposure induces inflammatory reaction. Fluoride exposure results changes in intestinal microbial abundance. Fluoride is an important trace element in the living body. A suitable amount of fluoride has a beneficial effect on
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Maturing of Purkinje cells in various metabolic blockades.
Abstract is not available
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Fluoride: Developmental Neurotoxicity.
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